Work

Decreased Hepatic Futile Cycling Compensates for Increased Glucose Disposal in the Pten Heterodeficient Mouse

Year: 2006

Type: article

Source: Diabetes

DOI: 10.2337/db06-0002

Authors: Jun Xu, Lori Gowen, Christian Raphalides, Katrina K. Hoyer, Jason G. Weinger, Mathilde Renard, Joshua J. Troke, Bhavapriya Vaitheesyaran, W.N. Paul Lee, Mohammed F. Saad, Mark W. Sleeman, Michael A. Teitell, Irwin J. Kurland

Institutions: Stony Brook University, Regeneron (United States), University of California, Los Angeles, State University of New York

Language: English

Topic: Metabolism, Diabetes, and Cancer

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: closed

Funders: National Cancer Institute, National Institute of Diabetes and Digestive and Kidney Diseases, National Center for Research Resources

Awards: A NOVEL MECHANISM OF TCL1 TUMORIGENESIS, G1984462341, G5433446791, Hepatic Insuling Action: Role of the Pentose Cycle, CRTC2 in Cellular Development, Function, and Neoplasia, CLINICAL NUTRITION RESEARCH UNIT, General Clinical Research Center, G7886326478, G796514787, PEROXISMAL AND MITOCHONDRIAL B-OXIDATION IN OBESITY

OpenAlex ID: W2154282229

FWCI: 0.756

Cites: 28

Cited by: 25

Related to: 10