Year: 2011
Type: article
Abstract: Neoplastic clone dominance is key to disease progression. In this issue of Blood , Fleischman et al suggest that in myeloproliferative neoplasms (MPNs), neoplastic clone dominance is linked to JAK2V617F effects on responsiveness to and production of tumor necrosis factor (TNF)–α.[1][1]
This
Source: Blood
Author: Hal E. Broxmeyer
Institution: Indiana University School of Medicine
Language: English
Subfield: Genetics (clinical)
Field: Medicine
Domain: Health Sciences
Open Access status: bronze
OpenAlex ID: W2157516374