Work

Excess cone cell proliferation due to lack of a functional NR2E3 causes retinal dysplasia and degeneration in rd7/rd7 mice

Year: 2001

Type: article

Source: Human Molecular Genetics

DOI: 10.1093/hmg/10.16.1619

Author: N. B. Haider

Institution: Jackson Laboratory

Language: English

Topic: Retinal Development and Disorders

Subfield: Ophthalmology

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: closed

Funders: National Institutes of Health, National Eye Institute, National Cancer Institute, Eunice Kennedy Shriver National Institute of Child Health and Human Development

Awards: G3549131314, G4753194685, Developmental Genetics, Retinal Disease: Molecular Basis and Pathophysiology, Cancer Center Support (CORE) Grant

OpenAlex ID: W2157773282

FWCI: 7.708

Cites: 29

Cited by: 203

Related to: 10