Work

Transgenic Overexpression of G5PR That Is Normally Augmented in Centrocytes Impairs the Enrichment of High-Affinity Antigen-Specific B Cells, Increases Peritoneal B-1a Cells, and Induces Autoimmunity in Aged Female Mice

Year: 2012

Type: article

Source: The Journal of Immunology

DOI: 10.4049/jimmunol.1102774

Authors: Masahiro Kitabatake, Teppei Toda, Kazuhiko Kuwahara, Hideya Igarashi, Mareki Ohtsuji, Hiromichi Tsurui, Sachiko Hirose, Nobuo Sakaguchi

Institutions: Kumamoto University, Juntendo University

Language: English

Topic: T-cell and B-cell Immunology

Subfield: Immunology

Field: Immunology and Microbiology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: closed

Funder: Japan Society for the Promotion of Science

Awards: Molecular mechanism of the selective AID targeting at the IgV-region, Epistatic interaction of FcgammaRIIB and Sle16 polymorphism in rheumatoid arthritis, The analysis of development of autoimmune disease and abnormalexpansion of B-1 cells in G5PR transgenic mice, Histological analysis of T cell differentiation process based on hypermulticolor-hypersensitive fluorescence imaging method, Regulation of HIV-1 restriction factor Apobec3G by mRNA export factor

OpenAlex ID: W2162133040

FWCI: 0.39

Cites: 36

Cited by: 13

Related to: 10