Work

Rapamycin Reverses Cellular Phenotypes and Enhances Mutant Protein Clearance in Hutchinson-Gilford Progeria Syndrome Cells

Year: 2011

Type: article

Source: Science Translational Medicine

DOI: 10.1126/scitranslmed.3002346

Authors: Kan Cao, John J. Graziotto, Cecilia D. Blair, Joseph R. Mazzulli, Michael R. Erdos, Dimitri Krainc, Francis S. Collins

Institutions: National Institutes of Health, National Human Genome Research Institute, University of Maryland, College Park, Harvard University, Massachusetts General Hospital

Language: English

Topic: Nuclear Structure and Function

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: closed

Funders: National Institutes of Health, National Institute on Aging, National Institute of Neurological Disorders and Stroke

Awards: G745814159, Transcriptional Regulation of PGC-1 in Huntington's Disease

OpenAlex ID: W2166403801

FWCI: 12.93

Cites: 38

Cited by: 350

Related to: 10