Work

Degradation by Cullin 3 and effect on WNK kinases suggest a role of KLHL2 in the pathogenesis of Familial Hyperkalemic Hypertension

Year: 2015

Type: article

Source: Biochemical and Biophysical Research Communications

DOI: 10.1016/j.bbrc.2015.11.067

Authors: Chong Zhang, Nicholas P. Meermeier, Andrew S. Terker, Katharina I. Blankenstein, Jeffrey D. Singer, Juliette Hadchouel, David H. Ellison, Chao-Ling Yang

Institutions: Shanghai Jiao Tong University, XinHua Hospital, Oregon Health & Science University, Charité – Universitätsmedizin Berlin, Portland State University, Sorbonne Paris Cité, Paris Cardiovascular Research Center, Portland VA Medical Center

Language: English

Topic: Ion Transport and Channel Regulation

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: green

Funders: U.S. Department of Veterans Affairs, National Natural Science Foundation of China, Shanghai Municipal Education Commission, National Institutes of Health, National Institute of Diabetes and Digestive and Kidney Diseases

Awards: G1297944576, G1387556593, G3230397741, Cullin3突变导致家族性高血钾型高血压的分子机制研究, Nephrology Training Grant, G9000193503, WNK Kinase Regulation of Thiazide-sensitive NaCl Transport

OpenAlex ID: W2176261762

FWCI: 0.724

Cites: 25

Cited by: 20

Related to: 10