Work

GRK2 compromises cardiomyocyte mitochondrial function by diminishing fatty acid-mediated oxygen consumption and increasing superoxide levels

Year: 2015

Type: article

Source: Journal of Molecular and Cellular Cardiology

DOI: 10.1016/j.yjmcc.2015.10.002

Authors: Priscila Y. Sato, J. Kurt Chuprun, Jessica Ibetti, Alessandro Cannavo, Konstantinos Drosatos, John W. Elrod, Walter J. Koch

Institution: Temple University

Language: English

Topic: Mitochondrial Function and Pathology

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: green

Funders: Temple University, National Heart, Lung, and Blood Institute, National Institute on Drug Abuse

Awards: Mechanisms of mitochondrial calcium exchange in heart failure, Novel Mechanisms of Cardiac Injury and Repair, Targeting GRK2 in the Heart, Mechanisms of Reduced Fatty Acid Oxidation and Cardiac Dysfunction in Sepsis, Dysregulation of metabolic and bioenergetic pathways by cocaine and HIV-1 in CNS, Targeting GRK2 (BARK1) in Heart Failure, Improving Cardiac Function After Myocardial Infarction, GRK and b-Arrestin Signaling in Cardiac Regeneration, Novel Mechanisms and Therapies in Heart Failure

OpenAlex ID: W2178720104

FWCI: 2.366

Cites: 15

Cited by: 62

Related to: 10