17-Allylamino-17-Demethoxygeldanamycin (17-AAG) abrogates the G 2 /M cell cycle checkpoint by down-regulating chk1 and selectively sensitizes tumor cells with defective p53 to topoisomerase I poison
Year: 2004
Type: conference-abstract
Source: Journal of Clinical Oncology
Authors: A. N. Tse, D. Solit, N. Rosen, G. Schwartz
Institution: Memorial Sloan Kettering Cancer Center
Language: English
Topic: Heat shock proteins research
Subfield: Molecular Biology
Domain: Life Sciences
Open Access status: closed
OpenAlex ID: W2278086980