Work

Protein kinase C-dependent activation of the tumor necrosis factor receptor-mediated extrinsic cell death pathway underlies enhanced apoptosis in human myeloid leukemia cells exposed to bryostatin 1 and flavopiridol.

Year: 2003

Type: article

Source: PubMed

Authors: Leanne Cartee, Sonia C Maggio, Rebecca Smith, Heidi M Sankala, Paul Dent, Steven Grant

Institutions: Virginia Commonwealth University, Virginia Commonwealth University Medical Center

Language: English

Topic: Cell death mechanisms and regulation

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: closed

Funders: National Institutes of Health, National Institute of Diabetes and Digestive and Kidney Diseases

Awards: PHASE 1 TRIAL--BRYOSTATIN 1 AND HIGH DOSE ARA C (HIDAC), POTENTIATION OF TAXANE ACTIVITY BY MEK/MAPK INHIBITORS, MECHANISTIC STUDIES OF PMA AND FLAVOPIRIDOL INTERACTIONS, Carcinoma cell radiosensitization by MAPK inhibition, Signaling Pathways Regulating Epithelial Cell Survival

OpenAlex ID: W2282189773

FWCI: 1.287

Cites: 70

Cited by: 21

Related to: 10