Work

Loss of muscle‐specific inositol phosphatase (MIP) and increased phosphatidylinositol 3,5‐bisphosphate (PI(3,5)P2) inhibit muscle contractility and produce a Brody's disease phenotype

Year: 2009

Type: article

Source: The FASEB Journal

DOI: 10.1096/fasebj.23.1_supplement.780.2

Authors: Thomas M. Nosek, Leticia Brotto, Cheng‐Kui Qu, Marco Brotto

Institutions: Case Western Reserve University, University of Missouri–Kansas City

Language: English

Topic: Muscle Physiology and Disorders

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: closed

Funders: American Heart Association, National Institutes of Health, National Heart, Lung, and Blood Institute

Awards: SHP-2 Inhibitors as Potential Therapeutic Agents for JMML and Noonan Syndrome, G634955885, G9000182998

OpenAlex ID: W2294491098

FWCI:

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