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Prdx1 deficiency in mice promotes tissue specific loss of heterozygosity mediated by deficiency in DNA repair and increased oxidative stress

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Year: 2012
Type: article
Source: Mutation research. Fundamental and molecular mechanisms of mutagenesis
DOI: 10.1016/j.mrfmmm.2012.04.004
Authors: Vamsi Rani, Carola A. Neumann, Changshun Shao, Jay A. Tischfield
Institutions: Rutgers, The State University of New Jersey, Medical University of South Carolina
Language: English

Topic: Redox biology and oxidative stress
Subfield: Molecular Biology
Field: Biochemistry, Genetics and Molecular Biology
Domain: Life Sciences
SDG: Good health and well-being

Open Access status: green

Funders: National Institutes of Health, National Cancer Institute, National Institute of Environmental Health Sciences
Awards: Mouse Model for Chemical and Radiation Mutagenesis, The Role of Peroxiredoxin1 & Reactive Oxygen Species in Breast Tumor Initiation, Role of Prdx 1 in Natural Killer Cells and Tumorigenesis, Rutgers Center for Environmental Exposure and Disease (CEED)
OpenAlex ID: W2014055138
FWCI: 0.749
Cites: 110
Cited by: 27
Related to: 10
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