Work

Reduction of Nuak1 Decreases Tau and Reverses Phenotypes in a Tauopathy Mouse Model

Year: 2016

Type: article

Source: Neuron

DOI: 10.1016/j.neuron.2016.09.022

Authors: Cristian A. Lasagna-Reeves, Maria de Haro, Shuang Hao, Jeehye Park, Maxime W.C. Rousseaux, Ismael Al-Ramahi, Paymaan Jafar-Nejad, Luis Vilanova-Velez, Lauren See, Antonia De Maio, Larissa Nitschke, Zhenyu Wu, Juan C. Troncoso, Thomas F. Westbrook, Jianrong Tang, Juan Botas, Huda Y. Zoghbi

Institutions: Baylor College of Medicine, Texas Children's Hospital, Johns Hopkins University, Johns Hopkins Medicine, Dan L Duncan Comprehensive Cancer Center, Howard Hughes Medical Institute

Language: English

Topic: Alzheimer's disease research and treatments

Subfield: Physiology

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: bronze

Funders: Howard Hughes Medical Institute, National Institute on Aging, National Institute of Neurological Disorders and Stroke, Eunice Kennedy Shriver National Institute of Child Health and Human Development

Awards: G369896221, G4444814008, Studying the Physiological Role of Nuak1 in Tau Pathogenesis, Morris K. Udall Centers of Excellence for Parkinson's Disease Research (P50), Johns Hopkins Alzheimer's Disease Research Center, Behavioral and cell-based complementary screens to identify druggable targets modulating tau protein levels

OpenAlex ID: W2529470828

FWCI: 7.292

Cites: 52

Cited by: 155

Related to: 10