Work

CaMKII is a nodal signal for multiple programmed cell death pathways in heart

Year: 2016

Type: article

Source: Journal of Molecular and Cellular Cardiology

DOI: 10.1016/j.yjmcc.2016.12.007

Authors: Ning Feng, Mark E. Anderson

Institutions: Johns Hopkins University, Johns Hopkins Medicine, Johns Hopkins Hospital

Language: English

Topic: Cell death mechanisms and regulation

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: green

Funders: National Institutes of Health, National Heart, Lung, and Blood Institute

Awards: G2805179092, G2896504358, G2915933487, G367708880, Autonomic Nerve Activity and Paroxysmal Atrial Fibrillation, Methionine sulfoxide reductase A and oxidized CaMKII in structural heart disease, G8629291795, G9000036974, G9000105951, G9000124480, G9000336094

OpenAlex ID: W2565584878

FWCI: 4.363

Cites: 82

Cited by: 107

Related to: 10