The perturbation, by aluminium, of receptor-generated calcium transients in hepatocytes is not due to effects of Ins(1,4,5)P3-stimulated Ca2+ release or Ins(1,4,5)P3 metabolism by the 5-phosphatase and 3-kinase
Year: 1990
Type: article
Abstract: Correspondence| September 15 1990 The perturbation, by aluminium, of receptor-generated calcium transients in hepatocytes is not due to effects of Ins(1,4,5)P3-stimulated Ca2+ release or Ins(1,4,5)P3 metabolism by the 5-phosphatase and 3-kinase S B Shears; S B Shears Search for other works by this author on: This Site PubMed Google Scholar A P Dawson; A P Dawson Search for other works by this author on: This Site PubMed Google Scholar J W Loomis-Husselbee; J W Loomis-Husselbee Search for other works by this author on: This Site PubMed Google Scholar P J Cullen P J Cullen Search for other works by this author on: This Site PubMed Google Scholar Biochem J (1990) 270 (3): 837. https://doi.org/10.1042/bj2700837a Views Icon Views Article contents Figures & tables Video Audio Supplementary Data Peer Review Share Icon Share Twitter LinkedIn Cite Icon Cite Get Permissions Citation S B Shears, A P Dawson, J W Loomis-Husselbee, P J Cullen; The perturbation, by aluminium, of receptor-generated calcium transients in hepatocytes is not due to effects of Ins(1,4,5)P3-stimulated Ca2+ release or Ins(1,4,5)P3 metabolism by the 5-phosphatase and 3-kinase. Biochem J 15 September 1990; 270 (3): 837. doi: https://doi.org/10.1042/bj2700837a Download citation file: Ris (Zotero) Reference Manager EasyBib Bookends Mendeley Papers EndNote RefWorks BibTex toolbar search Search Dropdown Menu toolbar search search input Search input auto suggest filter your search All ContentAll JournalsBiochemical Journal Search Advanced Search This content is only available as a PDF. © 1990 London: The Biochemical Society1990 Article PDF first page preview Close Modal You do not currently have access to this content.
Source: Biochemical Journal
DOI: 10.1042/bj2700837a
Language: English
Subfield: Plant Science
Domain: Life Sciences
Open Access status: bronze
OpenAlex ID: W2733588659