齧歯動物モデルでエンドトキシン誘導炎症はIL‐1a発現とCD45+白血球動員を上方調節し再上皮形成と創傷閉鎖の速度を上昇させる
Year: 2016
Type: article
Source: Wound Repair and Regeneration
Language: Japanese
Topic: Wound Healing and Treatments
Subfield: Rehabilitation
Field: Medicine
Domain: Health Sciences
Open Access status: closed
OpenAlex ID: W2743324008
FWCI:
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