Work

Proteomic Alterations Associated with Biomechanical Dysfunction are Early Processes in the Emilin1 Deficient Mouse Model of Aortic Valve Disease

Year: 2017

Type: article

Source: Annals of Biomedical Engineering

DOI: 10.1007/s10439-017-1899-0

Authors: P. M. Angel, D. A. Narmoneva, M. K. Sewell-Loftin, C. Munjal, L. Dupuis, B. J. Landis, A. Jegga, C. B. Kern, W. D. Merryman, H. S. Baldwin, G. M. Bressan, Robert B. Hinton

Institutions: Medical University of South Carolina, University of Cincinnati, Vanderbilt University, Cincinnati Children's Hospital Medical Center, Indiana University Indianapolis, University of Padua

Language: English

Topic: Cardiac Valve Diseases and Treatments

Subfield: Cardiology and Cardiovascular Medicine

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: green

Funders: Cincinnati Children’s Research Foundation, National Institutes of Health, National Heart, Lung, and Blood Institute, National Cancer Institute, National Institute of General Medical Sciences, National Center for Advancing Translational Sciences, Eunice Kennedy Shriver National Institute of Child Health and Human Development

Awards: Tie Tek Modulation of Cardiac Development, Angiogenesis due to Elastic Fiber Fragmentation in Early Aortic Valve Disease-Res, Indiana Pediatric Scientist Award(IPSA), Center for Clinical and Translational Science and Training, G6293820928, Developmental Determinants of Cardiovascular Disease, The Vanderbilt Institute for Clinical and Translational Research (VICTR), South Carolina COBRE in Oxidants, Redox Balance and Stress Signaling, Direct Tumor Glycan Profiling on Tissue Microarrays, Cincinnati Center for Clinical and Translational Sciences and Training, Vanderbilt Institute for Clinical and Translational Research (VICTR)

OpenAlex ID: W2747694221

FWCI: 1.265

Cites: 58

Cited by: 15

Related to: 10