Work

p66Shc regulates migration of castration-resistant prostate cancer cells

Year: 2018

Type: article

Source: Cellular Signalling

DOI: 10.1016/j.cellsig.2018.02.008

Authors: Matthew A. Ingersoll, Yu-Wei Chou, Jamie S. Lin, Ta-Chun Yuan, Dannah R. Miller, Yan Xie, Yaping Tu, Rebecca E. Oberley-Deegan, Surinder K. Batra, Ming-Fong Lin

Institutions: University of Nebraska Medical Center, Kaohsiung Chang Gung Memorial Hospital, The University of Texas MD Anderson Cancer Center, National Dong Hwa University, Creighton University, Kaohsiung Medical University

Language: English

Topic: Prostate Cancer Treatment and Research

Subfield: Pulmonary and Respiratory Medicine

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: bronze

Funders: U.S. Department of Defense, University of Nebraska Medical Center, ASN Foundation for Kidney Research, University of Nebraska Omaha, Ministry of Science and Technology, Taiwan, National Institutes of Health, College of Public Health, University of Nebraska Medical Center, Fred and Pamela Buffett Cancer Center, Kaohsiung Chang Gung Memorial Hospital, National Cancer Institute, National Institute of General Medical Sciences

Awards: G281700398, G3693820871, Nebraska Center for Cellular Signaling, G5247383749, G6612021484, G7493782163, The Study of New Pathogenic Mechanism of P66shc through Ezh2 in Hepatocellular Carcinoma, G8188792447, G9000167784, G9000320260, Buffett Cancer Center Support Grant, Cancer Biology Training Program

OpenAlex ID: W2789931394

FWCI: 2.028

Cites: 62

Cited by: 20

Related to: 10