Work

Nav1.1-Overexpressing Interneuron Transplants Restore Brain Rhythms and Cognition in a Mouse Model of Alzheimer’s Disease

Year: 2018

Type: article

Source: Neuron

DOI: 10.1016/j.neuron.2018.02.029

Authors: Magdalena Martinez-Losa, Tara E. Tracy, Keran Ma, Laure Verret, Alexandra Clemente-Perez, Abdullah S. Khan, Inma Cobos, Kaitlyn Ho, Li Gan, Lennart Mucke, Manuel Alvarez-Dolado, Jorge J. Palop

Institutions: Gladstone Institutes, University of California, San Francisco, Centro Andaluz de Biología Molecular y Medicina Regenerativa, University of California System, University of California, Los Angeles, Universidad Pablo de Olavide

Language: English

Topic: Alzheimer's disease research and treatments

Subfield: Physiology

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: bronze

Funders: Alzheimer's Association, S. D. Bechtel, Jr. Foundation and Stephen Bechtel Fund, BrightFocus Foundation, Gladstone Institutes, National Institutes of Health, Instituto de Salud Carlos III, National Institute on Aging, National Institute of Neurological Disorders and Stroke, National Center for Research Resources

Awards: Linking tau proteostasis with neuronal activity in FTD, Pathogenic events triggered by tau acetylation in neurodegenerative tauopathy, Behavioral Core, The Role of Cathepsin B and Cystatin C in Alzheimer's Disease, Restoring Brain Functions in Alzheimer Models with Interneuron Transplants, G41133098, SIRT1 Limits Microglial Toxicity in Alzheimer's Disease, Deficient KIBRA signaling at synapses in Alzheimer’s disease, Kinases Linking Abeta and Tau to Synaptic Dysfunction, Cell-Based Therapy to Restore Brain Functions in Mouse Models, G6522063230, EXTRAMULAR RESEARCH FACILITIES CONSTRUCTION, Tau acetylation in Alzheimer's disease, Molecular mechanisms in progranulin deficient frontotemporal dementia

OpenAlex ID: W2790954104

FWCI: 14.24

Cites: 64

Cited by: 237

Related to: 10