Injury, dysbiosis, and filaggrin deficiency drive skin inflammation through keratinocyte IL-1α release
Year: 2018
Type: article
Authors: Nathan K. Archer, Jay-Hyun Jo, Steven K. Lee, Dongwon Kim, Barbara Smith, Roger V. Ortines, Yu Wang, Mark C. Marchitto, Advaitaa Ravipati, Shuting S. Cai, Carly A. Dillen, Haiyun Liu, Robert J. Miller, Alyssa G. Ashbaugh, Angad S. Uppal, Michiko K. Oyoshi, Nidhi Malhotra, Sabine Hoff, Luis A. Garza, Heidi H. Kong, Julia A. Segre, Raif S. Geha, Lloyd S. Miller
Language: English
Subfield: Dermatology
Field: Medicine
Domain: Health Sciences
Open Access status: green
Funders: U.S. Department of Health and Human Services, Pfizer, Food Allergy Research and Education, Boehringer Ingelheim, Regeneron Pharmaceuticals, Moderna, National Institutes of Health, National Human Genome Research Institute, National Institute of Allergy and Infectious Diseases, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Center for Research Resources, National Center for Advancing Translational Sciences, NHLBI Division of Intramural Research
Awards: G1276288829, Request for Zeiss LSM 510 MP Multiphoton Imaging System, G1715099302, Human in vivo Th17 cell responses in cutaneous immunity to Staphylococcus aureus, STAT3-mediated immunity to Staphylococcus aureus in skin, G4074189266, G5692152927, The role of IL-1alpha in atopic dermatitis skin inflammation, G625104578, Mechanism and clinical pilot tst of volar fibroblast to create ectopic volar skin, G690901646, G8013535420, G9000171835, SEAL (Stopping Atopic dermatitis and ALlergy) Study: Prevent allergy by enhancing the skin barrier, Gene-Environment Interactions at the Skin Surface
OpenAlex ID: W2890876748