Work

Author Correction: Impaired perceptual learning in a mouse model of Fragile X syndrome is mediated by parvalbumin neuron dysfunction and is reversible

Year: 2018

Type: erratum

Source: Nature Neuroscience

DOI: 10.1038/s41593-018-0273-3

Authors: Anubhuti Goel, Daniel A. Cantu, Janna Guilfoyle, Gunvant R. Chaudhari, Aditi Newadkar, Barbara Todisco, Diego de Alba, Nazim Kourdougli, Lauren M. Schmitt, Ernest Pedapati, Craig A. Erickson, Carlos Portera-Cailliau

Institutions: University of California, Los Angeles, Cincinnati Children's Hospital Medical Center, University of Cincinnati

Language: English

Topic: Genetics and Neurodevelopmental Disorders

Subfield: Genetics

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: bronze

Funders: U.S. Department of Defense, Cincinnati Children's Hospital Medical Center, Simons Foundation Autism Research Initiative, National Institutes of Health, National Institute of Mental Health, National Institute of General Medical Sciences, National Center on Birth Defects and Developmental Disabilities, Eunice Kennedy Shriver National Institute of Child Health and Human Development, Medical Research and Materiel Command

Awards: G2040153736, G4119534169, G5850682332, MARC U*STAR Program at the University of California, Los Angeles, Mechanisms of neocortical and sensory hyperexcitability in Fragile X Syndrome, Anomalous Sensorimotor Physiology in Fragile X Syndrome, G8324221335

OpenAlex ID: W2901068560

Cites:

Cited by: 5

Related to: 10