Transferrin receptor 2 controls bone mass and pathological bone formation via BMP and Wnt signalling
Year: 2018
Type: article
Source: Nature Metabolism
Authors: Martina Rauner, Ulrike Baschant, Antonella Roetto, Rosa Maria Pellegrino, Sandra Rother, Juliane Salbach-Hirsch, Heike Weidner, Vera Hintze, Graeme Campbell, Andreas Petzold, Regis Lemaitre, Ian Henry, Teresita Bellido, Igor Theurl, Sandro Altamura, Silvia Colucci, Martina U. Muckenthaler, Georg Schett, Davide S. K. Komla-Ebri, J. H. Duncan Bassett, Graham R. Williams, Uwe Platzbecker, Lorenz C. Hofbauer
Institutions: Technische Universität Dresden, University of Turin, Max Bergmann Center of Biomaterials, Hamburg University of Technology, Biotechnologisches Zentrum, Max Planck Institute of Molecular Cell Biology and Genetics, Indiana University Indianapolis, Indiana University School of Medicine, Indiana University – Purdue University Indianapolis, Innsbruck Medical University, Universität Innsbruck, Heidelberg University, Friedrich-Alexander-Universität Erlangen-Nürnberg, Universitätsklinikum Erlangen, Imperial College London, University Hospital Leipzig, German Cancer Consortium (DKTK) Dresden, Center for Regenerative Therapies Dresden
Language: English
Topic: Bone Metabolism and Diseases
Subfield: Molecular Biology
Domain: Life Sciences
Open Access status: green
Awards: Glucocorticoid-induced Atrophy in Bone and Muscle, G1515516536, G1554667050, Origins of bone and cartilage disease., Cellular thyroid hormone availability: regulation of development and tissue repair, and pathogenesis of degenerative disease., Cellular thyroid hormone availability: regulation of development and tissue repair, and pathogenesis of degenerative disease., Cellular thyroid hormone availability: regulation of development and tissue repair, and pathogenesis of degenerative disease., Cellular thyroid hormone availability: regulation of development and tissue repair, and pathogenesis of degenerative disease., G726052161, G8992108187
OpenAlex ID: W2901508573