Work

Mitotic CDK Promotes Replisome Disassembly, Fork Breakage, and Complex DNA Rearrangements

Year: 2019

Type: article

Source: Molecular Cell

DOI: 10.1016/j.molcel.2018.12.021

Authors: Lin Deng, R. Alex Wu, Remi Sonneville, Olga V. Kochenova, Karim Labib, David Pellman, Johannes C. Walter

Institutions: Harvard University, Dana-Farber Cancer Institute, Dana-Farber/Harvard Cancer Center, University of Dundee, MRC Protein Phosphorylation and Ubiquitylation Unit, Broad Institute, Howard Hughes Medical Institute

Language: English

Topic: DNA Repair Mechanisms

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

Open Access status: bronze

Funders: Howard Hughes Medical Institute, American Cancer Society, Wellcome Trust, Cancer Research UK, National Institutes of Health, Medical Research Council, National Heart, Lung, and Blood Institute, National Cancer Institute, National Institute of General Medical Sciences

Awards: G1458394169, G1614540307, G2980745033, The mechanism of vertebrate DNA replication termination, Functional dissection of the eukaryotic replisome., G8441978230, Polyploidy, Aneuploidy and Genome Stability, Ubiquitylation pathways controlling the end of chromosome replication, Mechanism and regulation of DNA interstrand cross-link repair

OpenAlex ID: W2949368903

FWCI: 6.804

Cites: 80

Cited by: 154

Related to: 10