Work

Transcriptome signature of ventricular arrhythmia in dilated cardiomyopathy reveals increased fibrosis and activated TP53

Year: 2020

Type: article

Study design: Observational Study

Source: Journal of Molecular and Cellular Cardiology

DOI: 10.1016/j.yjmcc.2019.12.010

Authors: Mary E. Haywood, Andrea Cocciolo, Kadijah F. Porter, Evgenia Dobrinskikh, Dobromir Slavov, Sharon L. Graw, T. Brett Reece, Amrut V. Ambardekar, Michael R. Bristow, Luisa Mestroni, Matthew R.G. Taylor

Institutions: University of Colorado Anschutz, University of Colorado Denver, University of Colorado Hospital

Language: English

Topic: Cardiomyopathy and Myosin Studies

Subfield: Cardiology and Cardiovascular Medicine

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: green

Funders: Fondation Leducq, National Institutes of Health, National Heart, Lung, and Blood Institute, National Center for Advancing Translational Sciences

Awards: Whole genome DNA sequencing to identify novel genes in dilated cardiomyopathy, Colorado Clinical and Translational Science Institute (UL1), Genetics, Mechanisms and Clinical Phenotypes of Arrhythmogenic Cardiomyopathy, G4921130661, Molecular Genetics, Pathogenesis and Protein-Replacement Therapy in Arrhythmogenic Cardiomyopathy, Cardiomyocyte phenotype and mechanotransduction in Filamin C gene variants causing arrhythmogenic cardiomyopathy, Colorado Clinical and Translational Sciences Institute, Colorado Clinical and Transational Sciences Institute, Molecular Epidemiology of Dilated Cardiomyopath

OpenAlex ID: W2998798376

FWCI: 3.56

Cites: 69

Cited by: 23

Related to: 10