Work

HDAC6 inhibition promotes α-tubulin acetylation and ameliorates CMT2A peripheral neuropathy in mice

Year: 2020

Type: article

Source: Experimental Neurology

DOI: 10.1016/j.expneurol.2020.113281

Authors: Cristina Picci, Victor S.C. Wong, Christopher J. Costa, Marion C. McKinnon, David C. Goldberg, Michelle Swift, Nazia M. Alam, Glen T. Prusky, Sida Shen, Alan P. Kozikowski, Dianna E. Willis, Brett Langley

Institutions: Burke Medical Research Institute, Weill Cornell Medicine, University of Waikato, University of Illinois Chicago

Language: English

Topic: Hereditary Neurological Disorders

Subfield: Cellular and Molecular Neuroscience

Field: Neuroscience

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: closed

Funders: Dr. Miriam and Sheldon G. Adelson Medical Research Foundation, Burke Foundation, National Institutes of Health, National Institute of Neurological Disorders and Stroke

Award: Optimization of HDAC6 Inhibitors in the Treatment of CMT

OpenAlex ID: W3010355643

FWCI: 4.085

Cites: 53

Cited by: 63

Related to: 10