Copper is an essential regulator of the autophagic kinases ULK1/2 to drive lung adenocarcinoma
Year: 2020
Type: article
Source: Nature Cell Biology
Institution: University of Pennsylvania
Language: English
Subfield: Epidemiology
Field: Medicine
Domain: Health Sciences
Open Access status: green
Funders: U.S. Department of Health and Human Services, American Cancer Society, Pew Charitable Trusts, V Foundation for Cancer Research, University of Pennsylvania, Francis Crick Institute, National Institutes of Health, National Cancer Institute, National Institute of General Medical Sciences, National Institute of Environmental Health Sciences
Awards: G1370827282, G198386693, G3117330915, Translational Research Training Program in Environmental Health Sciences, G3373183056, G4211188739, G4577891079, Deconstructing the multi-faceted roles of Rb in tumor progression, G5988489043, Investigating paradoxical YAP activation as an emergent limitation to Cu chelation therapy in BRAF V600E-driven melanoma, G6995340829, G7423592327, Investigating the influence of copper on autophagy in pancreatic cancer, G7643702416, G8299400184, G8836997178, G9000235771, Center of Excellence in Environmental Toxicology, Molecular and Cellular Mechanisms of Copper-Dependent Nutrient Signaling and Metabolism, G947847320
OpenAlex ID: W3011699272