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Alternative splicing of APOBEC3D generates functional diversity and its role as a DNA mutator

Year: 2020
Type: article
DOI: 10.1007/s12185-020-02904-y
Authors: Hisashi Takei, Hirofumi Fukuda, Gilbert Pan, Hiroyuki Yamazaki, Tadahiko Matsumoto, Yasuhiro Kazuma, Masanori Fujii, Sohei Nakayama, Ikei S. Kobayashi, Keisuke Shindo, Riu Yamashita, Kotaro Shirakawa, Akifumi Takaori-Kondo, Susumu S. Kobayashi
Institutions: Beth Israel Deaconess Medical Center, Harvard University, Gunma University, Kyoto University, National Cancer Center Exploratory Oncology Research & Clinical Trial Center, National Cancer Center Hospital East, Harvard Stem Cell Institute
Language: English
Open Access status: green
Funders: American Cancer Society, National Institutes of Health, Harvard Stem Cell Institute, Japan Society for the Promotion of Science, National Cancer Institute, National Center for Chronic Disease Prevention and Health Promotion
Awards: Identification of the mechanisms underlying tumorigenesis by oncogenic tyrosine kinases, Elucidating the mechanisms of drug-tolerant cell formation in EGFR lung cancer, G2081208805, G2725395538, G2793864631, Overcoming resistance to tyrosine kinase inhibitors in lung cancer, APOBEC3B-induced genome mutations and its contribution to clonal evolution in cancers, Transcriptome analysis of HIV-1 latently infected cells, Emergence of drug resistant mutations in lung cancer
OpenAlex ID: W3035180457
FWCI: 0.243
Cites: 38
Cited by: 6
Related to: 10