Work

An Mb1-Cre-driven oncogenic Kras mutation results in a mouse model of T-acute lymphoblastic leukemia/lymphoma with short latency and high penetrance

Year: 2020

Type: article

Source: Leukemia

DOI: 10.1038/s41375-020-01036-w

Authors: Jacob J. Junco, Taylor Chen, Raushan Rashid, Maci Terrell, Vincent U. Gant, Matthew Miller, Rachel Rau, H. Daniel Lacorazza, Karen R. Rabin

Institution: Baylor College of Medicine

Language: English

Topic: Acute Lymphoblastic Leukemia research

Subfield: Public Health, Environmental and Occupational Health

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: green

Funders: U.S. Department of Health and Human Services, American Society of Hematology, Cancer Prevention and Research Institute of Texas, National Institutes of Health, National Cancer Institute, National Center for Research Resources

Awards: Molecular Epidemiology And Somatic Alterations Driving Acute Lymphoblastic Leukemia In Down Syndrome, BD Biosciences Special Order LSRII, G2859315832, G3102002109, G393611383, G5465708471, Mechanisms of KLF4 suppression in pediatric leukemia, Molecular epidemiology of acute lymphoblastic leukemia in children with Down syndrome, G7022586960, Advanced Multiparameter Cytometry and Cell Sorting Core, G7605004183, G7837447426, G8153776008, DEFINING THE ROLE OF DOT1L IN DNMT3A-MUTANT LEUKEMIA, G9000240210, Baylor College of Medicine Cancer Center - Cancer Center Support Grant

OpenAlex ID: W3085965519

FWCI: 0.374

Cites: 15

Cited by: 4

Related to: 10