Work

Young DAPK1 knockout mice have altered presynaptic function

Year: 2021

Type: article

Abstract: Young death-associated protein kinase 1 (DAPK1) knockout mice have reduced basal glutamate release probability, an effect that normalized with maturation. This provided a compensatory mechanism that may have prevented a reduction of long-term depression in the young DAPK1 knockout mice.

Source: Journal of Neurophysiology

DOI: 10.1152/jn.00055.2021

Authors: Dayton J. Goodell, Jonathan E. Tullis, K. Ulrich Bayer

Institution: University of Colorado Anschutz

Language: English

Topic: Neuroscience and Neuropharmacology Research

Subfield: Cellular and Molecular Neuroscience

Field: Neuroscience

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: green

Funders: National Institute of General Medical Sciences, National Institute of Neurological Disorders and Stroke