Reply to De Mey et al.
Year: 2022
Type: letter
Institutions: University of South Carolina, University of Nevada, Reno, Washington University in St. Louis, Augusta University, Loma Linda University, Medical College of Wisconsin, University of Virginia, University of New Mexico, University of Missouri, Nationwide Children's Hospital, Abigail Wexner Research Institute, The Ohio State University, Michigan State University
Language: English
Open Access status: green
Funders: Nationwide Children's Hospital, National Heart, Lung, and Blood Institute, National Institute of General Medical Sciences, National Institute of Diabetes and Digestive and Kidney Diseases, National Institute of Neurological Disorders and Stroke, National Institute of Environmental Health Sciences, National Institute of Biomedical Imaging and Bioengineering, Eunice Kennedy Shriver National Institute of Child Health and Human Development
Awards: G1264860245, Novel Non-Invasive Coronary Flow Patterning to Predict Early Coronary Microvascular Disease, Damage-Associated Molecular Patterns in Hypertension, Differential Macro- and Micro-Vascular Remodeling in Type 2 Diabetes and Metabolic Syndrome, G2274850893, G235079345, Structural Basis of Peripheral Coupling Sites Regulating Arterial Smooth Muscle Contractility (A1), Autophagy regulates β-hydroxybutyrate synthesis to prevent hypertension-associated premature vascular aging, G2549538869, G2578633385, G2578853757, TRP Channel-Dependent Regulation of Arterial Tone (Renewal), G4151364183, 5-HT7 receptor and blood pressure regulation, Mechanisms of Heterocellular Signaling at the Myoendothelial Junction, Mechanisms of Microvascular Remodeling Progression, Hydrogen sulfide regulation of vascular tone and blood pressure, G504730971, Regulation of Uterine Smooth Muscle Excitability, Role of ANO1 Channels and Its Regulation by PIP2 in EC-Coupling in Pulmonary Artery Myocytes, Novel Regulatory Mechanisms in the Human Microcirculation, Autophagy regulates β-hydroxybutyrate synthesis to prevent hypertension-associated premature vascular aging, Coordinating Unit for DiaComp, Intrarenal Arteries Sense N-formyl Peptides Leading to Vascular Injury in Sepsis, Maternal vascular responses to extracellular mitochondrial DNA during pregnancy, Sensory mechanisms in brain capillary endothelial cells that initiate functional hyperemia, Nevada Center of Biomedical Research Excellence in Molecular and Cellular Signal Transduction in the Cardiovascular System, Purinergic Regulation of Veinous Endothelial Permeability, Paracrine TNF signaling impairs endothelial TRPV4 microdomains in obesity, TRP channels as fundamental sensors of the cerebral microcirculation, A novel molecular mechanism for stimulating uterine contractility by oxytocin, Formyl peptide receptor activation induces vascular plasticity and remodeling inhypertension, Flow-Activated Calcium Signaling Microdomains in Small Pulmonary Arteries, Quantitative and computational characterization of oxytocin receptor signaling
OpenAlex ID: W4221133919