Testing the Effect of Internal Genes Derived from a Wild-Bird-Origin H9N2 Influenza A Virus on the Pathogenicity of an A/H7N9 Virus
Year: 2015
Type: article
Abstract: (Cell Reports 12, 1831–1841; September 22, 2015) In the originally published version of this article, the sentence “However, all but 2 (r1278 and r2578) of the 34 viruses containing SH09 PB1 replicated efficiently in eggs” should read “32 viruses” rather than “34 viruses.” In addition, the data for an additional five hybrid viruses in the “Virulence group II” section were omitted from Table 1, and the data in the “No. Seroconverted (HI Titers), Exposed” column and the “r2HA Q226L, G228S, PB2 E627K” row should read “3 (40, 20, 40)” rather than “3 (20, 40, 20)” in Table 2. In addition, the number “4” was omitted in the middle of the “Inoculated, p.i. Day” column in Table 2. The sentence has been revised and the complete tables now appear correctly online. The authors apologize for any confusion these errors may have caused. Testing the Effect of Internal Genes Derived from a Wild-Bird-Origin H9N2 Influenza A Virus on the Pathogenicity of an A/H7N9 VirusSu et al.Cell ReportsSeptember 3, 2015In BriefSu et al. characterize H7N9 reassortants bearing wild bird H9N2 virus genes and provide insight into the potential risk of the H7N9 avian influenza. An H7N9 virus acquires substantial virulence by reassorting with a wild bird H9N2 virus and airborne transmission by substituting amino acids associated with adaptation to mammals. Full-Text PDF Open Access
Source: Cell Reports
Language: English
Subfield: Epidemiology
Field: Medicine
Domain: Health Sciences
Open Access status: gold
Estimated APC paid: $5,620
OpenAlex ID: W4233609359