Work

Mitochondrial Antioxidant Function Is a Potential Mechanism for Organ Differences in Interleukin-1-Mediated Tolerance to Oxidative Injury

Year: 1999

Type: article

Source: The American Journal of the Medical Sciences

DOI: 10.1016/s0002-9629(15)40644-5

Authors: David M. Guidot, Andrew D. Kitilowski, Brooks M. Hyberston, John E. Repine

Institutions: Emory University, Atlanta VA Medical Center, University of Colorado Denver

Language: English

Topic: Mitochondrial Function and Pathology

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: closed

Funders: National Institutes of Health, National Heart, Lung, and Blood Institute

Awards: G1164335899, G2604400014, SCOR--OCCUPATIONAL AND IMMUNOLOGICAL LUNG DISEASES, MITOCHONDRIAL SUPEROXIDE GENERATION IN HYPEROXIA

OpenAlex ID: W1995470745

FWCI: 0.228

Cites: 30

Cited by: 4

Related to: 10