Work

Aldehyde dehydrogenase 3A1 protects airway epithelial cells from cigarette smoke-induced DNA damage and cytotoxicity

Year: 2013

Type: article

Source: Free Radical Biology and Medicine

DOI: 10.1016/j.freeradbiomed.2013.11.028

Authors: Jun-Ho Jang, Shannon Bruse, Yushi Liu, Veronica Duffy, Chunyu Zhang, Nathaniel Oyamada, Scott Randell, Akiko Matsumoto, David C. Thompson, Yong Lin, Vasilis Vasiliou, Yohannes Tesfaigzi, Toru Nyunoya

Institutions: Lovelace Respiratory Research Institute, University of New Mexico, New Mexico VA Health Care System, University of New Mexico Health Sciences Center, University of North Carolina at Chapel Hill, University of Colorado Denver

Language: English

Topic: Alcohol Consumption and Health Effects

Subfield: Pathology and Forensic Medicine

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: green

Funders: National Institutes of Health, National Institute on Aging, National Heart, Lung, and Blood Institute, National Eye Institute, National Cancer Institute, National Institute of Diabetes and Digestive and Kidney Diseases, National Institute of Environmental Health Sciences

Awards: G1321483612, G1903070866, G1917348475, G1997881128, Role and Molecular Mechanisms of Corneal Aldehyde Dehydrogenase, G3775936536, Regulation of Mucous Cell Metaplasia in Asthma, Resolution of Epithelial Cell Hyperplasia, G4945018210, G5219023017, G5580342918, G606193249, G6437100625, MOLECULAR MECHANISMS AND CORNEAL ALDEHYDE DEHYDROGENASE, University of Colorado Cancer Center Support Grant, UNC Cystic Fibrosis Research and Translation Core Center, Resolution of Epithelial Cell Hyperplasia in Asthma

OpenAlex ID: W1996723569

FWCI: 2.238

Cites: 36

Cited by: 76

Related to: 10