Work

N-glycan branching inhibits pro-inflammatory B cell activity by controlling TLR4 and BCR signaling.

Year: 2017

Type: conference-abstract

Source: The Journal of Immunology

DOI: 10.4049/jimmunol.198.supp.195.16

Authors: Christie-Lynn Mortales, Michael Demetriou

Institution: University of California, Irvine

Language: English

Topic: Glycosylation and Glycoproteins Research

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: closed

OpenAlex ID: W4313356154

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