Work

Loss of Abi1 promotes a non‐canonical Crk signaling, drives invasive cell phenotypes and co‐relates with poor patient survival in GBM

Year: 2016

Type: article

Source: The FASEB Journal

DOI: 10.1096/fasebj.30.1_supplement.1117.2

Authors: Sushil Kumar, Adam Sowalsky, Leszek Kotula, Raymond B Birge

Institutions: Rutgers, The State University of New Jersey, Beth Israel Deaconess Medical Center, Harvard University, SUNY Upstate Medical University

Language: English

Topic: Protein Kinase Regulation and GTPase Signaling

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: closed

Funder: National Institutes of Health

Awards: G2447841497, G6526714694

OpenAlex ID: W4389008018

FWCI:

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