Work

The mitochondrial redistribution of ENOS is regulated by AKT1 and dimer status

Year: 2024

Type: article

Source: Nitric Oxide

DOI: 10.1016/j.niox.2024.09.009

Authors: Xutong Sun, Santiago Moreno Caceres, Manivannan Yegambaram, Qing Lu, Marissa D. Pokharel, Jason T. Boehme, Sanjeev A. Datar, Saurabh Aggarwal, Ting Wang, Jeffrey R. Fineman, Stephen M. Black

Institutions: Florida International University, University of California, San Francisco

Language: English

Topic: Nitric Oxide and Endothelin Effects

Subfield: Physiology

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: green

Funders: National Heart, Lung, and Blood Institute, National Institute on Drug Abuse, National Human Genome Research Institute, National Institute of Environmental Health Sciences

Awards: Identification of AMD3100 (Plerixafor) as a lead compound for chlorine toxicity, Perinatal NO signaling in congenital heart disease, PKG Signaling and Sepsis Induced ALI, Genetics, Epigenetics, and Post-translational Modifications and the Development of Ventilator Induced Lung Injury (VILI), Metabolic reprogramming of smooth muscle cells in pulmonary hypertension, METABOLIC REPROGRAMMING AND PULMONARY VASCULAR DISEASE ASSOCIATED WITH CONGENITAL HEART DISEASE RESULTING IN INCREASED PULMONARY BLOOD FLOW, Chronic Widespread Pain in HIV: Novel Mechanisms and Therapeutics, Altered Lymphatic Function and Development in Congenital Heart Disease, FIU-Diversity Center for Genomic Research (FIU-DCGR)

OpenAlex ID: W4402829554

FWCI: 1.976

Cites: 75

Cited by: 7

Related to: 10