Work

Optimization of the Irf8 +32-kb enhancer disrupts dendritic cell lineage segregation

Year: 2024

Type: article

Source: Nature Immunology

DOI: 10.1038/s41590-024-01976-w

Authors: Feiya Ou, Tian-Tian Liu, Pritesh Desai, Stephen T. Ferris, Sunkyung Kim, Haolin Shen, Ray A. Ohara, Suin Jo, Jing Chen, J. Luke Postoak, Siling Du, Michael S. Diamond, Theresa L. Murphy, Kenneth M. Murphy

Institutions: Washington University in St. Louis, Saint Louis University, University of California, San Francisco

Language: English

Topic: Genomics and Chromatin Dynamics

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: green

Funders: Howard Hughes Medical Institute, U.S. Department of Health and Human Services, Cancer Research Institute, California Institute of Technology, Washington University in St. Louis, National Institutes of Health, School of Medicine, Stanford University, University of California, San Diego, National Heart, Lung, and Blood Institute, National Institute on Drug Abuse, National Cancer Institute, National Institute of Allergy and Infectious Diseases

Awards: G1886068547, G1927151046, G1938931754, G2354209684, G3014393894, G4563812999, Mechanism of c-MYC repression by IRF8 in myeloid lineages, G6503650262, Function of Wdfy4 in cross-presentation and immunity, G7234268382, G7660896353, Transcriptional basis of embryonic macrophage development, G8973680554, Understanding the Mechanisms of DC Licensing in CD8 T Cell Priming, Molecular Basis of cDC1 Development, G970892991

OpenAlex ID: W4403195453

FWCI: 1.553

Cites: 68

Cited by: 12

Related to: 10