Year: 2024
Type: article
Abstract: In this issue of Blood, Caruso and colleagues 1 provide compelling evidence that when the exceptional flexibility of erythrocytes is impaired, the resultant aberrant wall interactions during flow induce a molecular signal of endothelial inflammation that extends the classic model of vascular dysfunction in sickle cell disease (SCD).
Source: Blood
Author: Michael J. Simmonds
Institution: Griffith University
Language: English
Subfield: Genetics
Field: Medicine
Domain: Health Sciences
Open Access status: bronze
OpenAlex ID: W4404124078