Work

Canagliflozin treatment prevents follicular exhaustion and attenuates hallmarks of ovarian aging in genetically heterogenous mice

Year: 2024

Type: article

Source: GeroScience

DOI: 10.1007/s11357-024-01465-w

Authors: José V. V. Isola, Subhasri Biswas, Hashan Jayarathne, Chase R. Hubbart, Jessica D. Hense, Satoshi Matsuzaki, Michael T. Kinter, Kenneth M. Humphries, Sarah R. Ocañas, Marianna Sadagurski, Michael B. Stout

Institutions: Oklahoma Medical Research Foundation, Wayne State University, Oklahoma City VA Medical Center

Language: English

Topic: Reproductive Biology and Fertility

Subfield: Public Health, Environmental and Occupational Health

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: green

Funders: National Institutes of Health, National Institute on Aging, National Institute of General Medical Sciences, National Institute of Diabetes and Digestive and Kidney Diseases, National Institute of Environmental Health Sciences

Awards: Cellular senescence and epigenomic remodeling in ovarian aging, G4919595995, Canagliflozin as a Neuroprotective Agent to Improve Neuroinflammation and Cognitive Function during Aging, Molecular mechanisms underlying metabolic reprogramming by paternal benzene exposure, Michigan Diabetes Research Center (MDRC), IDeA National Resource for Quantitative Proteomics, Benzene exposure promotes neuroinflammation and metabolic dysregulation, Oklahoma Nathan Shock Center of Excellence in Basic Biology of Aging, Center for Cellular Metabolism Research in Oklahoma, Oklahoma IDeA Network of Biomedical Research Excellence

OpenAlex ID: W4405353940

FWCI: 5.362

Cites: 100

Cited by: 13

Related to: 10