Work

TMAO accelerates cellular aging by disrupting endoplasmic reticulum integrity and mitochondrial unfolded protein response

Year: 2025

Type: article

Source: Cellular and Molecular Life Sciences

DOI: 10.1007/s00018-024-05546-z

Authors: Fahimeh Varzideh, Emanuele Farroni, Urna Kaunsakar, Mahaba Eiwaz, Stanislovas S. Jankauskas, Gaetano Santulli

Institution: Albert Einstein College of Medicine

Language: English

Topic: Endoplasmic Reticulum Stress and Disease

Subfield: Cell Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: gold

Estimated APC paid: $4,290

Funders: American Heart Association, National Institutes of Health, National Heart, Lung, and Blood Institute, National Institute of Diabetes and Digestive and Kidney Diseases

Awards: Inositol 1,4,5-trisphosphate receptors and activation of cardiac fibroblasts in ischemia/reperfusion injury, Beta Cell Intracellular Calcium and Diabetes, A paradigm-shifting idea in storage-disease cardiomyopathy, G802470759

OpenAlex ID: W4406604830

FWCI: 2.51

Cites: 23

Cited by: 6

Related to: 10