Year: 2025
Type: article
Abstract: Cancer patients are at a four- to 6.5-fold higher risk of developing venous thromboembolic events (VTEs), with risk factors such as surgery, hospitalization, and chemotherapy. The pathophysiology of cancer-associated thrombosis (CAT) involves the direct or indirect activation of the coagulation cascade by the release of tumor-derived procoagulant factors (microparticles, tissue factors, cancer procoagulants, and platelet agonists). We have reviewed the published literature from the references on CAT.
Authors: Atul Laddu, Naha Sahithi, Siddharth Ranjan, Vedanshi Kumar, Ruth Bush
Institution: The University of Texas Medical Branch at Galveston
Language: English
Subfield: Internal Medicine
Field: Medicine
Domain: Health Sciences
Open Access status: gold
Estimated APC paid: $2,400
OpenAlex ID: W4407548369
