Work

Phosphomimetic Experiments Do Not Support a Causal Role for TFAM Phosphorylation in mtDNA Elimination in Sperm

Year: 2025

Type: article

Source: Journal of Molecular Biology

DOI: 10.1016/j.jmb.2025.169433

Authors: Natalya Kozhukhar, Yidong Bai, Mikhail F. Alexeyev

Institutions: University of South Alabama, The University of Texas at San Antonio Health Science Center, Mays Cancer Center at UT Health San Antonio

Language: English

Topic: Mitochondrial Function and Pathology

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: bronze

Funders: National Science Foundation, U.S. Department of Defense, National Institutes of Health, National Heart, Lung, and Blood Institute, NIH Office of the Director, National Cancer Institute, National Institute of Allergy and Infectious Diseases, Division of Cancer Epidemiology and Genetics, National Cancer Institute, Division of Microbiology and Infectious Diseases, National Institute of Allergy and Infectious Diseases

Awards: Characterization the disruption of mitochondrial function and induction of oxidative stress by SARS-CoV2, TOWARDS HUMANIZED MOUSE MODELS OF MITOCHONDRIAL DISEASE, Lung endothelial cell phenotypes, Seahorse XFe24 Extracellular Flux Analyzer, G2470163165, G304324391, G3674943084, G5075316425, Mouse models for mitochondrial disorders caused by mutations in mtDNA, Designer Mitochondria for Biotechnology, Healthcare, and Basic Research, G7278233107, The mitochondrial aspects of health disparity of hepatocellular carcinoma in Hispanic population

OpenAlex ID: W4414077910

FWCI: 0.505

Cites: 51

Cited by: 2

Related to: 10