Work

Dual-Input Regulation of β-Cell Proliferation by ATF6α and Glucose via E2F1

Year: 2025

Type: article

Source: Diabetes

DOI: 10.2337/db25-0279

Authors: Huguet V. Landa-Galvan, Thalia A. Castro, Jahi J. Noel, Gabriel Avila Llamas, Rohit B. Sharma, Laura C. Alonso

Institution: Weill Cornell Medicine

Language: English

Topic: Endoplasmic Reticulum Stress and Disease

Subfield: Cell Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: closed

Funders: National Institutes of Health, National Institute of Diabetes and Digestive and Kidney Diseases

Awards: Forced Activation of ATF6 Drives Pancreatic Beta Cell Dysfunction, ATF6 and the Beta Cell, Role of GRP78 in beta cell adaptation in obesity and diabetes, Diabetes Research and Training Center, Integrated Islet Distribution Program (U24) - 2021, Polyamines and Hypusine in Type 1 Diabetes Pathogenesis, Benefits and harms of activating ATF6 in beta cells, NYR-Diabetes Research Center (NYR-DRC)

OpenAlex ID: W4415773311

FWCI: 0.837

Cites: 45

Cited by: 2

Related to: