Work

The mechanism and targeted therapy of zinc mediating retinal ganglion cell injury through histone H4 deacetylation

Year: 2025

Type: conference-paper

Abstract: Objective: Mammalian optic nerve cannot regenerate once injured. Our previous study found that mobile zinc (Zn2+) increased rapidly after optic nerve injury and it comes from amacrine cell (AC). Whether chelating Zn2+ or specifically knocked out zinc transporter 3 (ZnT3) in ACs significantly [for full text, please go to the a.m. URL]

Source: German Medical Science (German Research Foundation)

DOI: 10.3205/24artvis09

Authors: Liu, Zhe, Tang, J.H., Zhang, Q., Liu, L.Y., Zhuo, Y.H., Li, Y.Q.

Institutions: Sun Yat-sen University, State Key Laboratory of Ophthalmology, Zhongshan Ophthalmic Center, Sun Yat-sen University

Language: English

Topic: Trace Elements in Health

Subfield: Nutrition and Dietetics

Field: Nursing

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: green

OpenAlex ID: W6889870761

FWCI:

Cites:

Cited by:

Related to: