Additional file 1 of Transcript profiling of plastid ferrochelatase two mutants reveals that chloroplast singlet oxygen signals lead to global changes in RNA profiles and are mediated by Plant U-Box 4
Year: 2025
Type: article
Abstract: Additional file 1: Supplemental information, Figures S1 through S15. Figure S1. Tetrapyrrole profiling of fc2 mutants, Figure S2. Visualization of variance among RNA-seq replicates used in the study, Figure S3. Volcano plot analysis of mean differential gene expression between genotypes in a single condition, Figure S4. Gene ontology analyses of differentially expressed genes in fc2 mutants, Figure S5. Analysis of stress marker gene expression in the single pub4 mutant, Figure S6. Gene ontology analyses of differentially expressed genes in fc2 pub4, Figure S7. WRKY, ERF, and NAC transcription factor networks are induced by chloroplast singlet oxygen accumulation, Figure S8. Gene Ontology term enrichment of differentially expressed transcription factors genes and transcription factor gene families. Figure S9, WRKY and NAC transcription factor targets are induced by chloroplast singlet oxygen accumulation and activation of NAC targets is blocked by the pub4 mutation. Figure S10, Gene ontology analyses of differentially expressed plastid protein-encoding genes in fc2 mutants, Figure S11. Gene ontology analyses of differentially expressed plastid protein-encoding genes shared between fc2 and fc2 pub4 mutants, Figure S12. Gene ontology analyses of differentially expressed plastid protein-encoding genes unique to fc2 pub4 mutants, Figure S13. Relative expression of plastid protein-encoding genes during singlet oxygen stress, Figure S14. Analysis of salicylic acid response marker gene expression in the single pub4 mutant, Figure S15. Activation of senescence pathways is blocked by the pub4 mutation.
Source: Figshare
Institutions: University of Arizona, University of California, Riverside
Language: English
Subfield: Molecular Biology
Domain: Life Sciences
Open Access status: green
OpenAlex ID: W6939576278
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