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C5aR1 deficiency in osteoblasts attenuates impaired bone fracture healing in lipopolysaccharide treated mice

Year: 2024

Type: conference-paper

Abstract: Objectives: In recent years evidence has accumulated that the complement system, a crucial part of innate immunity, is involved in bone turnover and the pathogenesis of inflammatory bone disorders. Our group showed that the anaphylatoxin receptor C5aR1 is upregulated during osteoblastogenesis, induces [for full text, please go to the a.m. URL]

Source: German Medical Science (German Research Foundation)

DOI: 10.3205/24dkou153

Authors: Bülow, Jasmin, Dieterich, Sandra, Krüger, Benjamin, Fischer, Verena, Haffner-Luntzer, Melanie, Huber-Lang, Markus, Relja, Borna, Ignatius, Anita

Institutions: Universität Ulm, University Hospital Ulm

Language: English

Topic: Bone Metabolism and Diseases

Subfield: Molecular Biology

Field: Biochemistry, Genetics and Molecular Biology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: green

OpenAlex ID: W6965081222

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