Work

CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer

Year: 2009

Type: article

Source: Nature Immunology

DOI: 10.1038/ni.1836

Authors: Cameron R Stewart, Lynda M Stuart, Kim Wilkinson, Janine M van Gils, Jiusheng Deng, Annett Halle, Katey J Rayner, Laurent Boyer, Ruiqin Zhong, William A Frazier, Adam Lacy-Hulbert, Joseph El Khoury, Douglas T Golenbock, Kathryn J Moore

Institutions: Harvard University, Massachusetts General Hospital, University of Massachusetts Chan Medical School, New York University, Washington University in St. Louis

Language: English

Topic: Immune Response and Inflammation

Subfield: Immunology

Field: Immunology and Microbiology

Domain: Life Sciences

SDG: Good health and well-being

Open Access status: green

Funders: Wellcome Trust, National Institute on Aging, National Institute of Allergy and Infectious Diseases, National Institute of Diabetes and Digestive and Kidney Diseases, National Institute of Neurological Disorders and Stroke

Awards: Mechanisms of CD36 Signal Transduction, The role of the phagosome in innate immune sensing, CD36 in Alzheimer's Disease, Toll-Like Receptor Signaling in Alzheimer's Disease, Defective clearance of dying cells, aberrant dendritic cell maturation and autoimmunity., Center for the Study of Inflammatory Bowel Disease at Massachusetts General Hospital

OpenAlex ID: W2045772297

FWCI: 32.25

Cites: 42

Cited by: 1,556

Related to: 10