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Thioredoxin reductase is required for the inactivation of tumor suppressor p53 and for apoptosis induced by endogenous electrophiles

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Year: 2006
Type: article
Source: Carcinogenesis
DOI: 10.1093/carcin/bgl111
Authors: P. B. Cassidy, K. Edes, C. C. Nelson, K. Parsawar, F.A. Fitzpatrick, P. J. Moos
Institutions: University of Utah, Huntsman Cancer Institute
Language: English

Topic: Redox biology and oxidative stress
Subfield: Molecular Biology
Field: Biochemistry, Genetics and Molecular Biology
Domain: Life Sciences
SDG: Good health and well-being

Open Access status: closed

Funders: National Institutes of Health, U.S. Public Health Service, National Cancer Institute, National Institute of Allergy and Infectious Diseases
Awards: Molecular and Clinical Approaches to Colon Cancer Precursors, The role of selenoproteins in the etiology of cancer, Leukotriene Biosynthesis: Inflammation and Cancer, G8100726529
OpenAlex ID: W2145402376
FWCI: 3.369
Cites: 60
Cited by: 106
Related to: 10
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