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Insulin Resistance and Ad-Hoc Hypotheses

Year: 2026

Type: article

Abstract: Type 2 diabetes is explained in the conventional model as a state of insulin resistancewith receptor defects, signaling pathway defects, and inflammation. This article argues thatinsulin resistance is not a signaling defect but a logistical problem: the glycogen storesare full. Insulin is a post-it note, glucose is distilled sunlight, and glycans are the body’slanguage. When one cuts away, the need for ad-hoc hypotheses disappears. Falsification issimple: fasting followed by a ketogenic diet normalizes all parameters. Type 2 diabetes isonly a chronic disease if one makes it so.

Source: Zenodo (CERN European Organization for Nuclear Research)

DOI: 10.5281/zenodo.19592494

Author: Lars H. Hasselby

Language: English

Topic: Diabetes Management and Research

Subfield: Endocrinology, Diabetes and Metabolism

Field: Medicine

Domain: Health Sciences

Open Access status: green

OpenAlex ID: W7154462027

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