Correction: The impact of inhibiting the Hippo signaling pathway effector molecule YAP1 on in vitro glioblastoma and glioblastoma stem cells
Year: 2026
Type: erratum
Abstract: The original version of this Article contained an error in Figure 6, in which the row containing the labels “Control, CA3, and siRNA” was positioned incorrectly. The original Figure 6 and accompanying legend appear below. Fig. 6 The alternative text for this image may have been generated using AI. The alternative text for this image may have been generated using AI. Full size image Assessment of cell migration by wound-healing (scratch) assay. (a–d) Representative phase-contrast micrographs acquired at 0, 24, and 48 h under vehicle control, CA3 (CIL56; IC₅₀), and YAP1-siRNA (optimized dose) in T98G, BCSC, BSC, and HBMEC cultures, respectively. Progressive closure of the linear scratch is used as a surrogate for collective cell migration. (e–h) Quantitative analysis of wound closure, presented as relative closure normalized to the control for each cell model. Bars denote mean ± SD from ≥ 3 independent experiments. Statistical comparisons were performed by one-way ANOVA with Tukey post hoc testing; significance indicated as **** p < 0.0001.
Source: Scientific Reports
Institutions: Ege University, Harran University, Muğla University
Language: English
Subfield: Cell Biology
Domain: Life Sciences
Open Access status: gold
Estimated APC paid: $2,690
OpenAlex ID: W7155105632