CRYAB is compromised in pathologically affected oligodendrocytes in multiple system atrophy
Year: 2026
Type: article
Abstract: CRYAB is hypermethylated and colocalizes with α-synuclein in multiple system atrophy oligodendrocytes.
Source: Brain Pathology
DOI: 10.1111/bpa.70112
Institutions: The University of Sydney, UNSW Sydney
Language: English
Subfield: Neurology
Field: Medicine
Domain: Health Sciences
Open Access status: gold
Estimated APC paid: $4,080
Funder: National Ataxia Foundation
Award: G7346066411
OpenAlex ID: W7162697539
